Glycation occurs when sugar molecules bond to proteins (like collagen), causing them to become stiff and dysfunctional (AGEs). Keeping HbA1c low prevents this structural damage to arteries, skin, and organs.

While CGM shows variability, HbA1c shows the 3-month average. It is the standard clinical metric for diagnosing diabetes and assessing long-term glycation damage to proteins.

Think of HbA1c as a measure of "rust" in your system. Every percentage point increase represents a significant increase in the glycosylation of vital proteins, leading to stiffer arteries, cataracts, and accelerated skin aging.

Glycation is the process where sugar bonds to protein, creating "sugar-coated" collagen that is stiff and yellow. High HbA1c is a marker of this accelerated aging process.

Keeping blood sugar low prevents the formation of "AGEs" (Advanced Glycation End-products), preserving the supple, youthful snap of the skin.